MUITO MAIS PERIGOSO QUE O ÉBOLA TRANSMISSÃO PODE DAR-SE APÓS 5 OU 10 MINUTOS DA AQUISIÇÃO OU SEJA QUANDO O COISO MEXE NA COISA ...SE A COISA TÁ INFECTADA É TIRO E QUEDA NÃO HÁ PERÍODO DE LATÊNCIA O COISO FICA APTO A TRANSPORTAR O VÍRUS E TRANSMITE-O DURANTE TRÊS DIAS ....UM FIM DE SEMANA NORMAL.... ah virus que s'apanham no bairro alto...Untersuchungen zur Resistenz von Beta-Rüben gegenüber Polymyxa betae : Vektory des beet necrotic yellow vein virus (BNYVV) Rhizomania disease of sugar beet (Beta vulgaris) is caused by beet necrotic yellow vein virus (FNYVV) (Tamada and Baba, 1973). The virus is transmitted to the roots of the plant by the soil-borne fungus Polymyxa betae (Keskin, 1964; Fujisawa and Sugimoto, 1977), The common name of the disease ('root madness') is derived from the root beard'ig symptom, caused by a proliferation of lateral rootlets. Above-ground symptoms are less specific; pale green leaves, often elongated and asymmetrical, with an upright posture. Leaves with typical yellow or necrotic veins are seldom observed and only occur when the virus is transported to the leaves. Rhizomania can cause a reduction of sugar content and root weight, which can result in sugar yield reductions of up to 80%. The host range of the virus is restricted to Beta spp. and spinach. The disease was first described in Italy (Canova, 1959), and later in Japan (Masuda et al., 1969). It is now widely distributed over most sugar beet growing areas in Europe (Asher, 1993) and the USA (Harveson and Rush, 1993). The occurrence in the Netherlands was confirmed in 1983 (Heijbroek, 1989). The virus survives in thick-walled resting spores of the vector. Both vector and virus can survive in soil for 15 years or more (Abe and Tamada, 1986). As a consequence of the slow decline of inoculum potential in soil (Schlösser, 1988), crop rotation adjustments cannot be applied as a control measure. A louder rotation can only h.lp to slow down the increase in infestation level. The damage by rhizomania can be reduced somewhat by agronomic measures that delay the time of initial infection to a later growth stage of the plant, e.g. early sowing (Ahrens, 1986) and transplanting of beet (Abe, 1987). Transplanting is too costly in European sugar beet growing, but it is applied in Japan (Abe, 1987). Soil disinfection was shown to increase yield (Schaufele, 1987), but is not an environmentally and economically desirable option for disease control. A number of chemical compounds were tested for their activity against the vector, but none effectively prevented infection of plants in the field (Schaufele, 1987; Asher, 1988). Biologia do BICHO NÃO TEM

afídeo alimenta-se e transmite virus durante dias 

o virus tem numerosos hospedeiros 

é como a pensão dos Loios....

qualquer um deles pode funcionar como repositorium da doença

o VAB VIRUS DO AMARELO DA BETERRABA 
É UM VIRUS SEMI-PERSISTENTE
QUE SE MANTEM NO VECTOR ENTRE 24 A 72 HORAS ....DISTÂNCIA DE 1 A 2 KM DO FOCO DE INFECÇÃO SERVE DE BARREIRA À DISPERSÃO DO VIRUS PELO COISO

PRIMEIROS SINTOMAS APARECEM NAS FOLHAS MAIS NOVAS ACLARAMENTOS OU MANCHAS CLORÓTICAS VULGUS CLOROSES DAS NERVURAS 

NA PÁGINA INFERIOR A NERVURA ENTRA EM DEPRESSÃO POR VEZES PROFUNDA 
É A CHAMADA DEPRESSÃO NERVOSA 

NERVURAS DEPRIMIDAS PELE DE CROCODILO PÕEM EM RELEVO ZONAS DO LIMBO E MESMO DO INFERNO....

AMARELECEM AS BASES DAS  FOLHAS MAIS VELHAS É COMO NA POLÍTICA PORTUGUESA ... 

Trichoderma harzianum was shown to infect resting 
spores of P. betae in laboratory experiments (D'Ambra and Mutto, 1986) and to reduce 
the severity of infection of beet roots by P. betae in naturally infested soil in greenhouse 
assays (D'Ambra et al., 1987; Camporota et al., 1988). However, as with other 
measures that result in a decrease of inoculum of the vector, the efficacy will be limited 
because the virus can still be transmitted by the fraction of resting spores that survive. 
The most promising solution for effective control of the disease is by breeding resistant 
cultivars (